RNASEH1 Chromosome 2

Ribonuclease H1
7 variants 7 Health Risk

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What This Gene Does
This gene encodes an endonuclease that specifically degrades the RNA of RNA-DNA hybrids and plays a key role in DNA replication and repair. Alternate in-frame start codon initiation results in the production of alternate isoforms that are directed to the mitochondria or to the nucleus. The production of the mitochondrial isoform is modulated by an upstream open reading frame (uORF). Mutations in this gene have been found in individuals with progressive external ophthalmoplegia with mitochondrial DNA deletions, autosomal recessive 2. Alternative splicing results in additional coding and non-coding transcript variants. Pseudogenes of this gene have been defined on chromosomes 2 and 17. [provided by RefSeq, Jul 2017]
Associated Conditions (5)
RNASEH1-related disorder
Inborn genetic diseases
Progressive external ophthalmoplegia with mitochondrial DNA deletions
autosomal recessive 2
Possible mitochondrial disorder - nuclear genes
Key Variants
All Variants (7)
RSID Category Clinical Significance Conditions
RS138292342 Health Risk Conflicting classifications of pathogenicity RNASEH1-related disorder, RNASEH1-related disorder
RS150583536 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS200495929 Health Risk Conflicting classifications of pathogenicity
RS1229409884 Health Risk Likely pathogenic
RS373442996 Health Risk Likely pathogenic Progressive external ophthalmoplegia with mitochondrial DNA deletions, autosomal recessive 2, Progressive external ophthalmoplegia with mitochondrial DNA deletions
RS1057517675 Health Risk Pathogenic Progressive external ophthalmoplegia with mitochondrial DNA deletions, autosomal recessive 2, Progressive external ophthalmoplegia with mitochondrial DNA deletions
RS766294940 Health Risk Pathogenic/Likely pathogenic Progressive external ophthalmoplegia with mitochondrial DNA deletions, autosomal recessive 2, Inborn genetic diseases
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