FAM111A Chromosome 11

FAM111 trypsin like peptidase A
24 variants 24 Health Risk

Upload your DNA to see your personal genotypes for variants in FAM111A.

What This Gene Does
The protein encoded by this gene is cell-cycle regulated, and has nuclear localization. The C-terminal half of the protein shares homology with trypsin-like peptidases and it contains a PCNA-interacting peptide (PIP) box, that is necessary for its co-localization with proliferating cell nuclear antigen (PCNA). Reduced expression of this gene resulted in DNA replication defects, consistent with the demonstrated role for this gene in Simian Virus 40 (SV40) viral replication. Mutations in this gene have been associated with Kenny-Caffey syndrome (KCS) type 2 and the more severe osteocraniostenosis (OCS, also known as Gracile Bone Dysplasia), both characterized by short stature, hypoparathyroidism, bone development abnormalities, and hypocalcemia. Alternative splicing results in multiple transcript variants. [provided by RefSeq, Aug 2015]
Associated Conditions (6)
FAM111A-related disorder
See cases
Inborn genetic diseases
Osteocraniostenosis
Autosomal dominant Kenny-Caffey syndrome
Skeletal dysplasia
Key Variants
All Variants (24)
RSID Category Clinical Significance Conditions
RS1298422871 Health Risk Conflicting classifications of pathogenicity FAM111A-related disorder, See cases, FAM111A-related disorder
RS140306279 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS142539088 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS147361273 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS149508004 Health Risk Conflicting classifications of pathogenicity FAM111A-related disorder, FAM111A-related disorder
RS187676082 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS2496296355 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Osteocraniostenosis, Autosomal dominant Kenny-Caffey syndrome
RS376721381 Health Risk Conflicting classifications of pathogenicity Skeletal dysplasia, Skeletal dysplasia
RS574553791 Health Risk Conflicting classifications of pathogenicity
RS587777012 Health Risk Conflicting classifications of pathogenicity Autosomal dominant Kenny-Caffey syndrome, Autosomal dominant Kenny-Caffey syndrome
RS748812251 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS749677037 Health Risk Conflicting classifications of pathogenicity
RS759624749 Health Risk Conflicting classifications of pathogenicity
RS761191576 Health Risk Conflicting classifications of pathogenicity
RS762116864 Health Risk Conflicting classifications of pathogenicity
RS778780878 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS944930983 Health Risk Conflicting classifications of pathogenicity Inborn genetic diseases, Inborn genetic diseases
RS145826377 Health Risk Likely pathogenic Osteocraniostenosis, Osteocraniostenosis
RS2135489647 Health Risk Likely pathogenic
RS587777015 Health Risk Pathogenic Osteocraniostenosis, Osteocraniostenosis
RS1565206032 Health Risk Pathogenic/Likely pathogenic Osteocraniostenosis, Autosomal dominant Kenny-Caffey syndrome, Osteocraniostenosis
RS587777011 Health Risk Pathogenic/Likely pathogenic Autosomal dominant Kenny-Caffey syndrome, Osteocraniostenosis, Inborn genetic diseases
RS587777013 Health Risk Pathogenic/Likely pathogenic Osteocraniostenosis, Osteocraniostenosis
RS587777014 Health Risk Pathogenic/Likely pathogenic Osteocraniostenosis, Osteocraniostenosis
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